Abstract : SCI includes both primary and secondary injuries, the former of which usually refers to a mechanical lesion to the spinal cord and the latter of which is caused by cellular and biological reactions to the main injury, including the vascular, neurological, and immunological systems. Oxidative stress is associated with SCI and can cause damage to cells. Repair enzymes such as SOD and quinine reductase play a crucial role in the repair and rebuilding process. Future studies on the possibility of tempol in the treatment of SCI hold a lot of promise.